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Furthermore, NT-enhanced collagen promoted fibroblast accumulation in granulation tissue and stimulated collagen production and deposition at the wound site, ultimately leading to a more organized and mature collagen matrix [197]

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2005).Taken together, these divergent findings underscore a critical and unresolved question: Can neurodegeneration proceed independently of BBB disruption, or is barrier compromise a central driver that facilitates the progression of neurodegenerative pathology