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This can lead to apoptosis (cell death), mitochondrial dysfunction, oxidative stress, and ultimately, cognitive impairment.[ii] The central treatment goals are to heal damaged neurons, restore neural communication and decrease the brains inflammatory status

Antioxidant defense mechanisms include both enzymatic (superoxide dismutase, catalase, glutathione peroxidase) and non-enzymatic systems

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Whether and how any of this translates into a meaningful outcome for a specific individual is exactly the kind of question that belongs in a clinical evaluation, not a generalized article

GPS has the ability to suppress the expression of these pro-inflammatory cytokines

When oxidative stress due to low glutathione interferes with cell communication and signaling in an autistic child's brain, the child will naturally suffer deficits in speech, learning, focus and other cognitive issues