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We have previously demonstrated that these fluxes of cytosolic glutamate can occur without significant increases in oxidative stress or altered cell viability (16)

Dual roles for HMGB1: DNA binding and cytokine

Selenium rescues orbital fibroblasts from cell death induced by hydrogen peroxide: Another molecular basis for the effects of selenium in graves orbitopathy

This process generates inflammation and oxidative stress

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Given the narrow range between inadequate and excessive intake, even minor variations may substantially affect the prevalence of thyroid dysfunction [17]