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doi:10.1021/ja01246a015

The findings have implications for the structural basis for the neuroprotective action of the ACore on full-length A-induced deficits in synaptic plasticity and fear memory (Forest et al., 2018)

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The precursor to TMAO, trimethylamine, on the other hand impairs BBB function, emphasizing the need to better understand the relationships between microbial-derived metabolites, host processing of these metabolites and BBB physiology [155]

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