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Further investigation showed that exposure of RAW264.7 cells to recombinant IFN-I, including IFN- and IFN-, activated the STAT-1 signaling pathway via phosphorylation of JAK1 and tyrosine kinase 2 (TYK2)

10.1016/j.bbi.2021.12.020 28 ChaM

Ultimately, rational design of targeted, context-specific, and combination-based interventions will be key to unlocking the full therapeutic potential of mTOR inhibition in both cancer and other diseases