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Distribution of glutathione and glutathione-related enzyme systems in mitochondria and cytosol of cultured cerebellar astrocytes and granule cells

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Therefore, regulation of GSH levels may be an effective approach to inhibit chondrocyte ferroptosis and may be one of the therapeutic strategies for OA

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These results show that the effect of inhibiting GSH synthesis acts on top of increased intracellular drug concentrations that are caused by ABCC1 loss and indicates that GSH is needed for efficient detoxification