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[79] However, the existence of many 4-hydroxycoumadin anticoagulants (for example phenprocoumon) that possess no ketone group in the 3-substituent to form such a structure, suggests that the hemiketal must tautomerise to the 4-hydroxy form in order for warfarin to be active

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Veto et al., 2010

Paradoxically, the expression of the corresponding histone methyltransferases MLL2 and G9a was increased in advanced atherosclerosis compared to early atherosclerosis [6]

Cell Therapy for Treatment of Intervertebral Disc Degeneration: A Systematic Review

10.1186/1744-8069-2-20 10 ChiuK.LauW