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One plausible explanation for these results is that the gpoA mutant is unable to effectively counteract the elevated ROS concentrations encountered within the intracellular environment of host cells, despite the partial compensatory activity of the TpxD and AhpD peroxidases (Fig

[7] [8] References [edit] 1 2 3 GRCh38: Ensembl release 89: ENSG00000243955 Ensembl, May 2017 1 2 3 GRCm38: Ensembl release 89: ENSMUSG00000057933 Ensembl, May 2017 "Human PubMed Reference:"

Walker, E

Arch Physiol Biochem 113:234258 Ghezzi P (2005) Regulation of protein function by glutathionylation

We assign this rate constant of the second phase to the intramolecular glutathione transfer within the roGFP2 WT -PfGrx fusion constructs