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Clin Gastroenterol Hepatol (2010) 8(2):10510

Conditional GPX4 knockout mice exemplify the risks of single-pathway targeting: while GPX4 deletion in PT cells protects against early AKI by eliminating damaged cells, long-term deletion activates the cGASSTING pathwaylikely via release of fragmented mitochondrial DNAleading to sustained type I interferon signaling and progressive interstitial fibrosis

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On day two, mice were placed into the same test box in the presence of two identical objects (6 cm away from the side wall) for 20 min, after an inter-trial interval of 1 h, one familiar object was replaced with a novel object

The ability of an antibody or a T-cell, specific for one antigen, to react with a second antigen