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This accumulation of the damage over time and across tissues is a primary risk factor in the genesis of many age-related diseases, such as cardiovascular disease (CVD), cancer, neurodegeneration, diabetes, osteoporosis, and sarcopenia [1, 2]

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Functional assays confirm that the overexpression of miR-144-5p enhances the protective effects of BMSC exosomes against GC apoptosis, whereas the inhibition of miR-144-5p reverses these benefits

The effect of matrix stiffness on the differentiation of mesenchymal stem cells in response to TGF-