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Description

Among the ACSL family members, ACSL4 preferentially catalyzes the formation of arachidonoyl-CoA (AA-CoA) by inserting coenzyme A (CoA) into arachidonic acid (AA), also called 5,8,11,14-eicosatetraenoic acid (20:4n-6, -6)

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This strategically shifts the detection window away from the highly positive regions where most common endogenous species oxidize, thereby securing the high selectivity required for reliable clinical diagnostics [170]

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Here several hypotheses are suggested, including persistent infections, reactivation of endogenous microbial reservoirs, infection-triggered autoimmunity, or other persisting post-infectious immune dysfunctions (Komaroff and Bateman, 2021

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X.ZhouY.ZhangC

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Isoforms changes of Tau protein during development in various species

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