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In this review, we synthesize the current understanding of how dysregulated copper metabolism and cuproptosis drive the pathogenesis of major cardiovascular conditions, including myocardial ischemia/reperfusion (I/R) injury, anthracycline-induced cardiotoxicity, atherosclerosis, diabetic cardiomyopathy (DCM), and sepsis-induced cardiac dysfunction, through pathways such as mitochondrial dysfunction, oxidative stress, and inflammation

For microscope examination at 400 magnification under a phase contrast microscope, 500 spermatozoa per group were classified as viable spermatozoa with intact acrosome stained with pink and non-viable spermatozoa with damaged acrosome stained with dark blue or lavender

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This process is called phase l and phase II glucuronidation