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Microenvironment-responsive phototherapeutic platforms that sense biochemical cues including redox status allow activation only within pathological niches, improving safety and enabling synergistic combinations with GSH-consuming or GSH-responsive agents to bias tumor redox balance toward therapeutic ROS generation [21]

For E1, the higher abundance of hydroxylated E1 also resulted in an increased abundance of 2- and 4-MeOE1, although these increases did not seem to be proportional to those observed for the hydroxylated metabolites

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This is due to the fact that alcohol acts both as a substrate and as an inhibitor of CYP2E1 (see Thummel et al

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