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Eligible trials were selected based on the inclusion criteria

(2021) found a metabolic switch toward mitochondrial -oxidation with an increased rate of mitochondrial respiration and a higher level of CPT2 in asthmatic bronchial SMC, whereas blocking CPT2 by either etomoxir or perhexiline drastically reduced the proliferation of asthmatic bronchial SMCs and remodeling in bronchial smooth muscles