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doi:10.1186/1742-2094-6-15

The etiology of PCOS involves a complex interplay of genetic, metabolic, hormonal, immunological and environmental factors, though its precise mechanisms remain incompletely understood

In contrast, Aur disrupts redox homeostasis by inhibiting thioredoxin reductase 1, leading to GSH and NADPH depletion, thereby amplifying ferroptotic signaling

We've built an open platform, and are continuing to open it further

8c) in mitochondria from PE placentae relative to PTC, suggesting that CER breakdown via ASAH2 is decreased in PE mitochondria leading to CER accumulation

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