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For example, Cu deficiency has been shown to promote the aberrant hydrophobicity of mutant SOD1, contributing to the impaired interaction of SOD1 and its neuronal toxicity, and this effect could be reversed by the supplementation of Cu

Estimation of blood-brain barrier crossing of drugs using molecular size and shape, and H-bonding descriptors

Studies demonstrate baicalein remarkably reduces neuronal apoptosis by downregulating the expression of pro-apoptotic proteins like Bax, cleaved caspase-3, and upregulating the anti-apoptotic Bcl-2 protein (Liu et al., 2018)

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