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Neuronal Slc25a39 deficiency abolished FGF21-mediated protection against mitochondrial damage and neuronal apoptosis following TBI in mice To elucidate the role of SLC25A39 in the biological protective effects of FGF21, we generated conditional knockout mice for Slc25a39 (Slc25a39 CKO ) by crossing Slc25a39 fl/fl mice with Map2-CreERT2 mice

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