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7 Discussions A growing body of evidence indicates that oxidative stress, mitochondrial dysfunction, and neuroinflammation are central to the pathophysiology of ASD

In patients with SLD, these integration sites predominantly occurred in intergenic regions (52.7%), followed by introns (43.7%) and exons (3.7%), which was consistent with observations in patients without SLD

Mechanisms: Activates AMPK, promoting fatty acid oxidation and glucose uptake while suppressing lipogenesis

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Statistical analyses were conducted using the GraphPad Prism 9.0 software suite