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Optimal exposure windows, dose scheduling, and the potential for hormetic effects or long-term consequences are poorly defined, impeding the rational design of exercise mimetic administration strategies

Together these results establish 4HNE as the biological ligand for GDAP1, provide mechanistic insight into 4HNE binding, and demonstrate that altered 4HNE recognition is the likely mechanism underlying CMT-causing mutants such as T157P near the 4HNE binding site

In vivo, PL-Au@P-ZnO NRs with US suppress tumor growth without causing systemic toxicity