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We can see a large number of articles using glutamate modeling to study the mechanism of excitotoxicity, most of which only look at the increase in calcium ions and mitochondrial dysfunction caused by excitotoxicity

In 2014, the US Food and Drug Administration (FDA) acknowledged a GRAS (Generally Recognised as Safe) notification for AOD 9604 as a food ingredient meaning the FDA raised no questions about the notifier's own safety conclusion

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In a study employing PC-12 cells, which are frequently used in neurobiological studies, the mechanism of toxicity revealed that Pb 2+ can trigger apoptosis through the altered ratio between the antiapoptotic protein BCl-2 and the proapoptotic proteins Bax and p53 in favor of their proapoptotic activity (Xu et al

Oxidative stress can damage GCs mitochondria, affecting mitochondrial energy metabolism and steroid hormone production