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2B), likely because of covalent binding of CAQK to plasma proteins, such as albumin, through the free sulfhydryl group (Pang et al, 2014) (Fig

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Thus, these NPs exhibit the potential for adjustable dosing in response to changing oxidative stress levels in AMD [120] (Table 3)

Two of the five littermates were Aldh2 / /Adh5 / mice (two females), as confirmed by genotyping, and three other animals were Aldh2 / /Adh5 +/ (male and female) and Aldh2 / /Adh5 +/+ (male) mice (Fig