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Kinetics and mechanism of protein tyrosine phosphatase 1B inactivation by acrolein Chem

Current hypotheses propose mechanisms involving ecological dysbiosis, intestinal barrier disruption, abnormal enterohepatic circulation and BA metabolism, and the translocation of microbial-derived metabolites (Figure 2)

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Handbook of Microalgal Culture

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Regarding cellular mechanisms, disruption of autophagic flux in AD results in the buildup of autophagic vesicles filled with undigested matter within dystrophic neurites