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By enhancing the mechanisms of mitochondrial quality control, UA effectively addresses a fundamental defect observed in cardiometabolic diseases: the persistent presence of dysfunctional mitochondria that exacerbate oxidative stress

Ingram DA, Lien IZ, Mead LE, et al

C3b binding to diamide-treated RBC was about two orders of magnitude higher than that of anti-band 3 NAbs and even occurred under alternative complement pathway conditions (Lutz et al., 1987)