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[109] Some epidemiological studies have suggested possible increased risks for specific congenital malformations, particularly cardiac defects and orofacial clefts, though these findings have not been consistently replicated across all research populations

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These pathway details matter in research because carnitine status can vary by species, tissue, diet, cell type, and experimental design

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For instance, physical changes like arthritis may restrict motion, which in turn may lead to a decline in assessment [9]

Gene products at the transcriptional and post-transcriptional levels, in addition to the mutation itself, can reveal the underlying pathophysiological processes of PD (Tnges et al., 2022)