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Modulation of oxidative stress, inflammation, and atherosclerosis by lipoprotein-associated phospholipase A2

The primary analysis utilized symptoms reported within the past 6 months of survey completion

Then, the PCR reactions were conducted using a previously described protocol 41

MicroRNA-216a inhibits neuronal apoptosis in a cellular Parkinsons disease model by targeting Bax

Notably, addition of hemin or pyruvate, a source for NAD+ did not impact these growth phenotypes, suggesting that amelioration of heme defect or respiration inhibition is not sufficient to overcome anti-proliferative effects of mitochondrial GSH depletion (Extended Data Fig