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This is in qualitative agreement with our previous study using the same culture and CSE exposure approach where CS-induced changes were much more drastic on the transcript than on the protein level 30 (Online Supplement 2: Final Protein List)

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Cysteine oxidation and disulfide formation in the ribosomal exit tunnel

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Hepatocytes have been considered as one of the origins of ICC, however, the underlying mechanisms remain unclear

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