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Chen, W., Dong, B., Liu, W

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Indeed, Sp was able to restore cytosolic and mitochondrial redox balance in neuronal cell line SHSY-5Y subjected to 1-methyl-4-phenylpyridinium (MPP + ) treatment which is known to cause mitochondria dysfunction and oxidative stress through complex I inhibition 1 (Fig

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Due to its metabolite (acetaminophen), phenacetin has a long duration of action

A BMP-controlled metabolic/epigenetic signaling cascade directs midfacial morphogenesis