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Description

Birkenfeld AL, Shulman GI

They found differential profiles across formulations, with the formulation designed to target immune modulation yielding the greatest reduction in IFN- secretion in cells challenged with IL-2

Since GSH interacts with the 1st glutathione-interaction site, glutathionylated products of the random bi-uni mechanism might already adopt a suitable orientation as classical GSSR substrates for the subsequent deglutathionylation (Fig