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Under normal physiological conditions, neuronal action potentials are transmitted to presynaptic terminals, causing voltage-gated Ca 2+ channels to open and extracellular Ca 2+ to flow in, prompting synaptic vesicles to release glutamate into the synaptic cleft, where glutamate binds to glutamate receptors on the postsynaptic membrane, triggering the influx of cations into the postsynaptic neuron and altering the membrane potential to achieve signal transmission

A proposed mechanism for autism: an aberrant neuroimmune response manifested as a psychiatric disorder

Porphyromonas gingivalis triggers inflammation in hepatocyte depend on ferroptosis via activating the NF-B signaling pathway

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The metabolic state-driven is typically based on metabolome profiling/metabolic state to characterize the antibiotic-resistant metabolome/metabolic state, identify putative metabolic mechanisms underlying antibiotic-resistance, and select critical nutrient metabolites as metabolic reprogramming agents 10,11,12

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