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The treatment option for AILI is limited, focusing mainly on APAP-induced mitochondrial oxidative stress

Accumulated evidence indicates that the initiation and progression of CIPN are tightly related with the impairment of chemotherapeutic agent-induced intraepidermal nerve fibers (IENF) [1], oxidative stress [2], abnormal spontaneous discharge, ion channel activation [3], the up-regulation of various pro-inflammatory cytokines, and the activation of the neuro-immune system [4, 5] (Fig

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These models significantly improve diagnostic accuracy yet still face challenges in interpretability [198]

it then activates the downstream MAPKK, and finally, MAPKK will activate the most downstream MAPK, this cascade reaction allows the signal to be amplified and transmitted within the cell (Jonak, 2002)