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This nanoagent could be decomposed by GSH to create Mn 2+ and GSSG, accompanied by the elimination of intracellular GSH (Figure 1F)

6) may serve as sensitive readouts of antioxidant adaptation in disease-relevant models

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(10 M in serum-free DMEM) increases intracellular glutathione, protecting hypoxic PC12 cells (a model for brain ischemia) from cell death [39]

Yang and co-workers reported a nanomedicine composed of dendritic mesoporous silica nanoparticles (DMSNs), Fe 3 O 4 , Mn, and glutaminase inhibitor Telaglenastat (CB-839) (termed DFMC) for restored oxaliplatin chemosensitivity on the basis of the GSH-responsive and -exhausting effect (Figure 4C) (Wu et al., 2022

Chen et al