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In models of inflammatory bowel disease, baicalin restores tight junction expression and reduces mucosal permeability, consistent with modulation of barrier injury driven by cytokine-induced EMT

S-nitrosylation of endothelial nitric oxide synthase is associated with monomerization and decreased enzyme activity

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Finally, we highlight emerging therapeutic strategies aimed at rewiring TAM metabolism or exploiting ferroptosis to overcome immune suppression and therapy resistance