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Similarly, the mouse model where -catenin was selectively deleted in Wnt-responsive cells in the dorsal part of the otocyst shortly before canal pouch evagination displayed the failure of formation of two of the SSCs

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Rg1 can effectively alleviate PD-associated motor and cognitive dysfunctions through various mechanisms, including the inhibition of neuroinflammation, reduction of oxidative stress, promotion of neuronal survival, and enhancement of synaptic plasticity