glutathione levels in breast cancer cells

glutathione levels in breast cancer cells (GSH) Human Cell Lines Exposed for 24 h to Each Unveiling the Role of Glutathione

Name: glutathione levels in breast cancer cells (GSH) Human Cell Lines Exposed for 24 h to Each Unveiling the Role of Glutathione
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Unveiling the Role of Glutathione S transferase Protein in Breast Cancer Drug Resistance: Insights into the Signaling Pathways and Therapeutic Implications ACS Pharmacology & Translational Science Glutathione and Xanthine Metabolic Changes in Tamoxifen Resistant Breast Cancer Cell Lines are Mediated by Down Regulation of GSS and XDH and Correlated to Poor Prognosis Adipocyte derived glutathione promotes obesity related breast cancer by regulating the SCARB2 ARF1 mTORC1 complex: Cell Metabolism Involvement of glutathione peroxidases in the occurrence and development of breast cancers Journal of Translational Medicine Springer Nature Link The crosstalk between glutathione metabolism and non coding RNAs in cancer progression and treatment resistance ScienceDirect Glutathione peroxidase 4 inhibition induces ferroptosis and mTOR pathway suppression in thyroid cancer Scientific Reports

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Description

As postulated by Zechmann (2020) and Otulak-Kozie et al

Notably, canonical GPX4 inhibitors, such as RSL3 and ML162, also inhibit thioredoxin reductase 1 (TXNRD1) 121,122

Since GSH is the final product of the homocysteine metabolism in the transsulfuration pathway by transferring sulfur from homocysteine to cysteine, a deficiency in transsulfuration pathway leads to excessive homocysteine production (hyperhomocysteinemia) and reduced GSH synthesis (159, 160)

Caught in the cROSsfire: GSH controls T cell metabolic reprogramming

Ten-eleven Translocation (TET) enzymes converts methylcytosine (m 5 C) to hydroxymethylcytosine (hmC), which is not recognized by Dnmt1, it is believed to exclude maintenance methylation allowing for passive demethylation, and it is an intermediate in the active conversion of m 5 C to cytosine (C) 27