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136 Since EAAT2 plays a central role in synaptic plasticity and neurotransmission, disruption of the metabolic and structural complexes in microdomains that enable EAAT2 clearance of glutamate suggests a role for atypical transporter localization in the pathophysiology of brain diseases

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4b) [68, 70, 71, 83, 105]

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The glutamine synthetase (GLUL) gene promoter is demethylated and thymine DNA glycosylase expression is elevated as a consequence of Myc-induced thymine DNA glycosylase upregulation [23, 43]

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