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Surfaceome profiling of rhabdomyosarcoma reveals B7-H3 as a mediator of immune evasion

September 2003 Sompayrac, LM

The pathophysiological basis of these disorders indicates the overproduction of superoxide anions, mitochondrial dysfunction, and altered signalling transduction that with damages DNA, proteins, or lipids resulting in inflammation and cell death

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conducted a randomized crossover trial of a modified ketogenic diet in the treatment of Alzheimers disease

Li et al., 2018), where it exerts its neuroprotective effects primarily by inhibiting the expression of pro-apoptotic factors such as Bax, caspase-3, and caspase-12, while promoting the expression of anti-apoptotic factors Bcl-2 and Bcl-xL