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(10) examined gene expression in vascular smooth muscle cells to study the complex interaction between oxidative injury and the pathogenesis of vascular disease

However, fibroblasts with the PSEN1 mutation did not show an increased colocalization of their mitochondria with p62/SQSTM1 compared to controls (Figure 4C)

Similar content being viewed by others Introduction Psoriasis vulgaris is a chronic immune-mediated inflammatory skin disease characterized by scaly skin plaques caused by keratinocyte hyperproliferation and immune cell infiltration 1

PiZZ, protease inhibitor ZZ phenotype

The goal is to restore levels to the upper-normal range, not to achieve supraphysiological levels

Statements Author contributions LF-G: Conceptualization, Data curation, Investigation, Methodology, Supervision, Validation, Visualization, Writing original draft, Writing review and editing