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The two forms of autophagy-related cell death are not independent of each other and can be triggered simultaneously, and cell death processes may switch between these two modalities under certain conditions [26]

At the immune level, metabolic crosstalk critically shapes polarization: persistent long-chain fatty acid (LCFA) exposure depletes intestinal SCFAs while promoting differentiation of nave T cells into pro-inflammatory Th1/Th17 subsets in the small intestine (Haghikia et al., 2015)