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For most cells, access to selenium is easy because there are not many enzymes that require selenium as a building block, and so the synthesis of antioxidative stress enzymes and molecules proceeds unimpeded

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Nonetheless, with the exception of certain viral encephalitis and prion disorders [499], such conditions are not fundamentally enigmatic and are less central to current neurodegeneration-focused drug development efforts compared to diseases like AD or frontotemporal lobar degeneration

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Figure 1 illustrates the alteration in glycolysis, beta-oxidation, and the metabolism of ketone body and asparagine in GDM

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