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Description

While GLP-1 therapies work through incretin pathways, cagrilintide activates amylin receptors located in key metabolic control centers of the brain[2]

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Further research is needed to fully understand its potential therapeutic applications and any associated risks

The result tempers claims that DSIP functions as a cortisol-lowering 'anti-stress' peptide in humans and underscores how limited and inconsistent the controlled human endocrine data for DSIP remain