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Exposure to pro-oxidant stimuli usually induces Nrf2 activation and upregulation of antioxidant enzyme expression through binding to the antioxidant response element (ARE), localized in the antioxidant enzyme gene promoters

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As we observe that STIM2, Orai1, and Orai3 proteins expressions are reduced by this treatment, reduced coupling between STIM2 and Orai more likely explains the reduced Ca 2+ entry after P 2 Y receptors activation than an inhibition of Ca 2+ release from the ER

sFASL binds to the Fas receptor (CD95) on the surface of target cells, activating the caspase cascade and triggering programmed cell death

[DOI] [PubMed] [Google Scholar] 237.Ezaki O

This interaction is critical in breast cancer oncogenesis and progression, particularly as Bag-1 activity is enhanced by mutant p53, further promoting cell survival and resistance to apoptosis ( Figure 3 ) (57, 58)