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The N-methyl-d-aspartate (NMDA) receptor-mediated excitatory procedure, which provides the intracellular Ca2+ increase necessary for nNOS activation, is another mechanism by which UCB-induced neuronal damage occurs

[77] [78] Other NAD-dependent enzymes include bacterial DNA ligases, which join two DNA ends by using NAD + as a substrate to donate an adenosine monophosphate (AMP) moiety to the 5' phosphate of one DNA end

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In this review, we emphasized the latest progress in understanding the mechanisms of IGF in acute and chronic pain, focusing on how IGF affects pain through changes in neuronal excitability, neuroinflammation, and glial cells

Regulation of GSK3/Nrf2 signaling pathway modulated erastin-induced ferroptosis in breast cancer

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