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10.1038/nrn3346 52 CryanJ

In early-stage PD patients, somatic mtDNA point mutations are markedly elevated in SN dopaminergic neurons, with respiratory chain defects resulting from mtDNA deletions being particularly prominent (117, 118)

Even without the preadministration of ABXs, when mice were simultaneously administered with APAP and hABXs (APAP/hABXs (0 h)), APAP-ALI did not occur in APAP mice (Fig

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