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Cao, J., Belousoff, M

Laboratory studies using rodent models demonstrate activation of neurons within hindbrain structures such as the area postrema and nucleus tractus solitarius following amylin receptor stimulation [1]

Then, confronted with obstructed vessels, there is circumvention of the occlusion, which may be the particular action of BPC 157 in ischemia/reperfusion

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Independent work has reported acceleration of tendon-to-bone healing in rotator cuff models in rats [Chang et al., Journal of Applied Physiology, 2011]