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Therefore, before considering iron chelators as disease-modifying therapies for neurodegenerative disorders requiring lifelong treatment, longer-term clinical studies are necessary to establish systematic safety monitoring protocols and to carefully weigh the potential benefits of reducing cerebral iron load against the risks associated with chronic administration

A molecular target for viral killer toxin: TOK1 potassium channels

Diabetic hyperglycaemia activates CaMKII and arrhythmias by O-linked glycosylation

Pea albumin attenuates dextran sulfate sodium-induced colitis by regulating NF-kappaB signaling and the intestinal microbiota in mice

10.1016/j.yhbeh.2014.12.007 Horm