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Collectively, these findings identify CD36-driven lipid peroxidation as a mechanistic link between exogenous lipid availability, ferroptosis susceptibility, and T-cell dysfunction in CRC

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Intracellular ROS generation visualized using the fluorescent probe H2DCFDA by fluorescence microscopy showed that total ROS levels were increased at 90 mM NaCl and substantially elevated at 120 mM NaCl (Figure 2F)