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(2013) developed an international clinical practice guideline for prevention and treatment of acute and late radiation reactions

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Additionally, pinpointing genetic risk factors for ME/CFS will help combat the disbelief some health professionals have towards ME/CFS ( There are also genetic predispositions that render certain individuals susceptible to developing autoimmune diseases (83) and even after Epstein-Barr virus (EBV) infection (84), such as human leukocyte antigen (HLA) variants (84, 85) that have experienced selective pressures whilst co-evolving with pathogens (86, 87)

For a subset of genes, AR binding by low doses of doxorubicin increased at sites that harbored binding motifs for prostate-specific factors, such as FOXA1, supporting that AR dependence on cofactors at half sites may be the basis for differential modulation by doxorubicin [67]

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