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Proximal tubules (high energy demand and mitochondria-rich) rely on FAO for ATP ( 3.2.5 Ferroptosis modulators in AKI-to-CKD intervention Ferroptosis modulators ( Table 1 ) target ferroptosis components to prevent AKI-to-CKD transition

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reported that the relative release of glutamate was inhibited by the knockout of alpha-synuclein in a mouse model in SN terminals (Watson et al., 2009)