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We previously demonstrated that an intracellular GPCR, TAAR1, was required for RhoA activation after AMPH treatment [3]

The analysis indicated that N,N-Diethylethanolamine was elevated in the aqueous humor of glaucoma patients and thus a biomarker, but we suspected that it was derived from oxybuprocaine (Supplementary Fig

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Mitochondrial function: NAD+ supports mitochondrial health and biogenesis while glutathione protects mitochondrial membranes from peroxidation damage, ensuring adequate energy supply for cellular repair processes

A combination of GalN/LPS has been widely used to induce ALF in animal models