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(2) p53 triggers transcription-independent apoptosis by binding directly to antiapoptotic Bcl-2 at the site of the BH3 binding pocket

Juarez VM, Montalbine AN, Singh A

this leads to an increase in ribosomal collisions, and premature termination of translation as well as UPS-dependent clearance of the N-terminal GPX4 fragment [96]

Assessing individual metabolic responsiveness to a lipid challenge using a targeted metabolomic approach

Its small size and positive charge make it an effective tool for studying mitochondrial stability under stress

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